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Procedure Practice 12/15/97 - Coding Recommendations

   
Feature Article 12/15/97:
Cerebrovascular Diseases
   

With its effects upon the brain, heart, kidneys, and extremities, vascular disease is the leading cause of morbidity and mortality in the United States. It is no wonder that vascular diseases are familiar to coders. We spend hundreds of hours every year coding vascular conditions, diagnostic, and therapeutic procedures. This month, we are reviewing some of the most common cerebrovascular diseases, along with the procedures done to diagnose and treat them. It is important to note here that, because of the prevalence of vascular diseases and the need for effective treatment, diagnostic and therapeutic methods are constantly evolving. Coders should communicate regularly with their vascular specialists to keep up with the latest changes.
   

Major Arteries of the Head and Neck

When blood leaves the heart via the left ventricle, it enters the aorta, the central artery in the body from which all arteries branch. The aorta is generally divided into three parts: the ascending aorta, the aortic arch, and the descending aorta. The ascending aorta is the shortest part with only two branches, the right and left coronary arteries, which supply blood to the heart muscle. As the aorta leaves the pericardial sac, it arches dorsally and to the left, forming the aortic arch. Three branches arise from the arch: the brachiocephalic artery, the left common carotid artery, and the left subclavian artery. These arteries supply blood to the head, neck, and arms.

The brachiocephalic artery travels only a short distance before it divides into the right subclavian artery and the right common carotid artery. Together with the left common carotid artery, which arises directly from the aortic arch, the right common carotid artery supplies most of the blood to the head and neck. The common carotids travel up the neck alongside the trachea and then divide again into the internal carotid artery and the external carotid artery. At the point where the common carotids divide (also called the bifurcation), the vessels enlarge, forming what is called the carotid sinus. This sinus has pressure receptors to aid in blood pressure control. In the same area is a small oval carotid body containing chemoreceptors which monitor changes in oxygen, carbon dioxide, and pH levels in blood traveling to the brain. The external carotid carries blood to most of the head and neck except for the brain. The internal carotid artery supplies the brain through its end branches, the anterior and middle cerebral arteries.

The right and left vertebral arteries branch off the subclavian artery, traveling upward and providing another major blood supply to the brain. They pass up through the transverse foramina of the cervical vertebrae. Once in the cranial vault, they unite to form the basilar artery.
   

Diseases of the Cerebrovascular System

Acute organic conditions affecting the cerebrovascular system include arteriosclerosis, occlusion, thrombosis, and hemorrhage. Cerebrovascular disease is grouped into one of five categories depending upon the patient’s presenting symptoms:

  1. Asymptomatic Disease: An audible bruit may be heard in the neck as the only manifestation of any underlying disease.
      
  2. Transient Neurologic or Visual Deficit: Symptoms in this category depend upon where an embolus is lodged, the size and composition of the embolus, and the presence of collateral vessels.
       
  3. Acute Unstable Neurologic Deficit: Patients have increasing transient ischemic attacks (TIAs), stroke in evolution, or waxing and waning neurologic symptoms. Urgent treatment is required.
       
  4. Completed Stroke: Patients are still at risk for another stroke without immediate intervention.
       
  5. Vertebrobasilar Disease: Emboli or hypoperfusion of the vertebral and basilar arteries which supply blood to the back of the brain can cause drop attacks, clumsiness, or other sensory problems.
       

Arteriosclerosis and Atherosclerosis

Arteriosclerosis is a chronic, generalized condition occurring in small arterioles as well as in large vessels. During the aging process, fibrosis and some intimal thickening develop, with weakening and disruption of the elasticity of the walls of the great arteries (e.g., the aorta and its major branches). The smooth muscle layer of the vessels atrophies to a certain extent, and the lumen of the aorta or one of its branches widens (ectasia) and may lead to aneurysm.

Atherosclerosis affects medium and large arteries and is characterized by patchy intramural thickening that encroaches on the arterial lumen, and in its most severe form causes obstruction. The atherosclerotic plaque consists of accumulated lipids and other cellular debris.

Plaque may grow slowly over years and produce severe stenosis or total occlusion. With time, the plaque becomes calcified and may undergo spontaneous rupture into the bloodstream. The ruptured plaque stimulates thrombosis; the thrombi may embolize, rapidly block the lumen, or gradually become incorporated into the plaque, contributing to its bulk and occlusive properties.
   

Carotid Artery Occlusive Disease

Atherosclerotic carotid artery disease is the leading cause of stroke. Treatment is aimed at prevention of this complication. "Mini-Strokes" or TIAs don't always precede a permanent stroke, and severe blockage (>70%) is usually treated surgically, even if there have been no symptoms.
   

Transient Ischemic Attacks

Transient ischemic attacks (TIAs) are one of the earliest signs of cerebrovascular disease. These episodes may last only seconds or as long as 24 hours. Some of the neurologic deficits during a TIA are identical to those seen with stroke, but they are of short duration with no lingering effects. Symptoms may include temporary paralysis of a limb, inability to speak or garbled speech, or changes in vision. Amaurosis fugax (temporary blindness of one eye) is a term commonly seen in a description of presenting symptoms in TIA patients. The etiology of a TIA is temporary obstruction of a cerebral vessel by a small particle of atheromatous debris or a blood clot usually embolized from plaque in the carotid artery.
   

Stroke in Evolution and Completed Stroke

A stroke in evolution is a growing infarction of brain tissue with neurologic symptoms that increase over a period of 24 to 48 hours. A completed stroke is an infarction of brain tissue manifested by neurologic deficits of varying degrees.

Stroke is responsible for nearly 150,000 U.S. deaths each year and ranks as the third leading killer in the United States. A stroke (or cerebrovascular accident) occurs when a vessel which supplies blood to the brain is blocked by an intraluminal blood clot (cerebral thrombosis) or when it ruptures (cerebral hemorrhage). Flow of blood to the brain is reduced, causing the area of affected brain tissue to degenerate and die. The area of tissue breakdown is called an infarct or infarction. A cerebral hemorrhage is a much more serious type of stroke because the blood escapes into brain tissue under high pressure, thus causing more extensive damage.

Stroke may also be caused by disease in any of the precerebral arteries arising from the aorta to supply the brain. As mentioned above, one common site is at the origin of the internal carotid artery in the neck, where atheromatous plaques may narrow the lumen and reduce blood flow. These plaques may also become ulcerated, and thrombi may form on the roughened surfaces. Bits of debris or thrombus may break off and travel to the brain where they block smaller cerebral arteries. Once a thrombus breaks off and travels through the vascular system it is called an embolus. Sometimes the internal carotid artery becomes completely blocked by a thrombus before it breaks apart, leading to a large cerebral infarction.
   

Subclavian Steal Syndrome

Subclavian steal is another condition that causes reduction in the amount of blood reaching the brain. As noted above, the vertebral arteries feed the back of the brain, and branch off the subclavian artery. In subclavian steal, blockage of the subclavian artery occurs proximal to the vertebral artery branch. This blockage causes reversal of blood flow through the vertebral artery as the subclavian artery steals blood that normally flows up to the brain.
   

Cerebral Aneurysm

An aneurysm is a localized dilation of an artery due to weakness of the arterial wall. Aneurysms may occur in any of the arteries that supply the brain. Occasionally an aneurysm occurs in the large cerebral arteries at the base of the brain. The most common type is a congenital cerebral aneurysm. Over time, the weakness of the artery causes a bubble or saclike protrusion of the artery through the defect. Though the weakness is present from birth, the aneurysm does not usually develop until adulthood. Cerebral aneurysms larger than 2.5 cm in diameter are termed giant. Their incidence is around 5% of all intracranial aneurysms. The peak age of occurrence is 60 years. The danger, as with any aneurysm, is that the aneurysm sac will rupture and cause tissue damage. When this happens in the brain, the results can be fatal.
   

Diagnostic and Therapeutic Procedures

Some of the following tests are used to diagnose not only cerebrovascular disease, but vascular lesions of other circulatory sites as well. A review of the results of these tests in the medical record is important for correct code assignment.

  • Cerebral Angiogram/Cerebral Arteriogram: This test tracks cerebral blood flow by injecting radiopaque dye into the carotid and vertebral arteries. The course of the dye is followed by serial x-rays.
      
  • Digital Subtraction Angiography (DSA): DSA is also an x-ray study of the carotid artery. It is similar to arteriography except that less dye is used.
       
  • Magnetic Resonance Angiography (MRA): This is one of the newest imaging techniques. It is more accurate than ultrasound and avoids the risks associated with x-rays and dye injection. An MRA is a type of magnetic resonance image that uses special software to create an image of the arteries in the brain. The shifts in the magnetic field where blood flows through vessels allows three-dimensional "mapping" of arteries and veins. Although the results of this test are not as accurate as a traditional contrast arteriogram, MRAs do not require the injection of contrast dye and therefore involve less risk for the patient.
       
  • Doppler Ultrasound Imaging: This is a highly versatile, noninvasive test in which sound waves are bounced off soft tissue. Ultrasound gives anatomic and physiologic information regarding the location and severity of blockages in arteries and veins. Similar to radar, scanners can detect the turbulent, high velocity flow patterns common around serious arterial narrowing. The degree of stenosis is usually expressed as a percentage of the normal diameter of the vessel opening.
       
  • Plethysmography: This method of detecting vessel blockages senses the quantity and quality of arterial blood flow into an extremity. Coupled with the measurement of segmental blood pressures (four to six separate measurements in each extremity), plethysmography accurately predicts the location and significance of arterial disease.
       
  • Oculoplethysmography (OPG): This procedure measures the pulsation of the arteries in the back of the eye. It is used as an indirect check for blockages in the carotid arteries.
       
  • Spiral Computed Tomography: With sophisticated changes to a decades-old technology, SPECT can produce 3-dimensional views of the arterial and venous systems. This technology is not often needed, but provides important information when it is used.
       
  • Transcutaneous Oximetry: Since the skin is the last part of an extremity to receive blood, the amount of oxygen reaching the surface of the skin is an important clue to the perfusion of the underlying bone and muscle. When needed, small sensors are placed on the extremity to measure the actual oxygen content of the skin.
       
  • Laser Doppler Flowmetry: A laser-beam and tiny air bladder measure the blood pressure in the skin capillaries. Like transcutaneous oximetry, this test is not often needed, but is used to predict if a wound can heal without major surgery, or if only a vascular reconstruction will provide sufficient blood and oxygen.
       
  • Angioscopy: A fiberoptic catheter is passed directly into the artery to visualize the blockage or damage or to assess the success of a repair. This is sometimes used as a substitute to a contrast arteriogram for patients allergic to contrast dyes.
       
  • Carotid Endarterectomy: A surgical excision of the arteriosclerotic lining of the carotid artery increases the caliber of the lumen and improve blood flow through the vessel. This procedure is often accompanied by a carotid artery bypass graft. The graft may be either synthetic, e.g., polytetrafluoroethylene (PTFE), or autologous, with vessel harvested from either the saphenous or jugular vein.
       
  • Percutaneous Balloon Angioplasty and Stenting: This is a relatively new procedure for carotid artery disease, though balloon angioplasty has been done for coronary arteries for some time. In this procedure, a balloon-tipped catheter is maneuvered to the site of carotid artery obstruction, and then the plaque causing the obstruction is pushed to the sides of the artery as the balloon is expanded. Sometimes a cage or wire stent is then inserted into the carotid to support the work done by the balloon and maintain vessel patency.
       
  • Aneurysm Clipping and Occlusion: Aneurysms are difficult to treat and are attacked in several ways. This method involves applying a stainless steel clip to the neck of the aneurysm to effectively seal it off from its blood supply. Once this is done, the aneurysm is usually deflated by the surgeon. A postoperative angiogram is done to verify obliteration of the aneurysm. All modern aneurysm surgery is performed with the aid of the operating microscope and micro-instrumentation.
       
  • Endovascular Aneurysm Coil Occlusion: This procedure is often done following an incomplete clipping of an aneurysm when residual aneurysm remains. The surgeon inserts a coil within the vessel to complete the obliteration of the aneurysm.

Now that you have a basic understanding of the cerebrovascular system’s structure and various pathologies, try your hand at coding the operative procedure on our Procedure Practice page. Assign the ICD-9-CM diagnosis code and the appropriate ICD-9-CM and CPT-4 codes for the surgical procedure. Our suggestions appear on our Coding Recommendations page.

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Major Arteries of the Head and Neck
Diseases of the Cerebrovascular System
Diagnostic and Therapeutic Procedures


     

Bibliography - References:
Allen, P.J. et al. "Saphenous vein versus PTFE carotid patch angioplasty," American Journal of Surgery, August 1997, pp. 73-75.
Crowley, Leonard V. MD. Introduction to Human Disease, third edition, copyright 1992.
MacDonald, J.D. "Recent advances in neurovascular surgery," Current Opinion in Neurology, February 1997, pp. 73-75.
Merck Manual, 16th Edition, copyright 1992, Merck and Company, Whitehouse Station, NJ
NYU Department of Neurosurgery website: URL:http://mcns10.med.nyu.edu
Society of Cardiovascular and Interventional Radiology et al, Interventional Radiology Coding Users’ Guide, Fourth edition, copyright 1995.
Spence, Alexander P. and Elliott, Mason. Human Anatomy and Physiology, 2nd edition, copyright 1983.
Thielen, K. et al. "Endovascular treatment of cerebral aneurysms following incomplete clipping," Journal of Neurosurgery August 1997, pp. 184-189.
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